eosin y solution Search Results


eosin y  (Chem Impex International)
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Chem Impex International eosin y
Eosin Y, supplied by Chem Impex International, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y right supplemental information eosin y disodium salt  (Valiant Co Ltd)
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Eosin Y Right Supplemental Information Eosin Y Disodium Salt, supplied by Valiant Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin-y solution  (Labonord SAS)
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Eosin Y Solution, supplied by Labonord SAS, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin-y solution  (Cellpath Inc)
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Cellpath Inc eosin-y solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution, supplied by Cellpath Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y solution  (Merck KGaA)
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Merck KGaA eosin y solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution, supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin 1  (Morphisto GmbH)
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Morphisto GmbH eosin 1
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin 1, supplied by Morphisto GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y solution  (Fisher Scientific)
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Fisher Scientific eosin y solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution, supplied by Fisher Scientific, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y solution 058-00062  (FUJIFILM)
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FUJIFILM eosin y solution 058-00062
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution 058 00062, supplied by FUJIFILM, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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1 % eosin solution  (Carl Roth GmbH)
90
Carl Roth GmbH 1 % eosin solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
1 % Eosin Solution, supplied by Carl Roth GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y solution  (Daejung Chemicals)
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Daejung Chemicals eosin y solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution, supplied by Daejung Chemicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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eosin y solution  (Beijing Solarbio Science)
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Beijing Solarbio Science eosin y solution
PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
Eosin Y Solution, supplied by Beijing Solarbio Science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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0.5% eosin y ethanol solution  (FUJIFILM)
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PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and <t>eosin</t> staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.
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Image Search Results


PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and eosin staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.

Journal: Journal of Cell Science

Article Title: The receptor protein tyrosine phosphatase PTPRK promotes intestinal repair and catalysis-independent tumour suppression

doi: 10.1242/jcs.261914

Figure Lengend Snippet: PTPRK supports recovery from damage and suppresses tumour growth and invasion in vivo . (A) Schematic of the CRISPR-targeted 22 bp deletion in Ptprk exon 1 to disrupt expression of PTPRK. The start ATG codon is underlined. (B) Immunoblot analysis of colon tissue lysates from Ptprk +/+ , Ptprk +/− and Ptprk −/− mice using antibodies recognizing PTPRK in addition to related members of the R2B family of RPTPs: PTPRM (also known as PTPµ) and PTPRU (also known as PTPϕ). Molecular masses are shown in kDa. Blots shown are representative of three mice. (C) DSS treatment regimen and Kaplan–Meier curves for untreated Ptprk +/+ mice ( n =5), and DSS-treated Ptprk +/+ ( n =8) and Ptprk −/− ( n =8) mice over the 14-day experiment. Endpoints as described in the Materials and Methods. Log-rank (Mantel–Cox) test. (D) Colon lengths of mice measured on day 7 following DSS-induced colitis. Ptprk +/+ , n =3; Ptprk −/− , n =3. Mean±s.d. Unpaired, two-tailed t -test. (E) AOM–DSS colorectal cancer model treatment regimen. (F) Individual tumour sizes from each Ptprk +/+ ( n =12) and Ptprk −/− ( n =11) mouse colon. Mean±s.e.m. Unpaired, two-tailed t -test. (G) Representative images of Ptprk −/− mouse colorectal tumour that has invaded into submucosa (indicated by white arrowheads). Haematoxylin and eosin staning. Scale bars: 200 μm. (H) Graph of percentage of mouse colons displaying tumour invasion into submucosa. Ptprk +/+ , n =12; Ptprk −/− , n =11. Analyses in F–H were conducted by a pathologist who was unaware of the treatment groups.

Article Snippet: Lastly, the slides were counterstained in Eosin-Y solution (Cell path, RBC-0100-00A) for 30 s. The sections were then dehydrated through 95% alcohol, absolute alcohol and xylene.

Techniques: In Vivo, CRISPR, Expressing, Western Blot, Two Tailed Test